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Liang, W. H., Xiao, C. W., & Oset, E. (2013). Study of eta K(K)over-bar and eta ' K(K)over-bar with the fixed center approximation to Faddeev equations. Phys. Rev. D, 88(11), 114024–10pp.
Abstract: In the present work we investigate the three-body systems of eta K (K) over bar and eta'K (K) over bar, by taking the fixed center approximation to Faddeev equations. We find a clear and stable resonance structure around 1490 MeV in the squared eta K (K) over bar scattering amplitude, which is not sensitive to the renormalization parameters. Conversely, we get only an enhancement effect of the threshold in the eta'K (K) over bar amplitude that indicates the difficulty to bind the eta'K (K) over bar system as a consequence of the eta'K interaction being weaker than the eta K one. We associate the eta K (K) over bar state found to the eta(1475).
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ATLAS Collaboration(Aad, G. et al), Cabrera Urban, S., Castillo Gimenez, V., Costa, M. J., Fassi, F., Ferrer, A., et al. (2013). Search for long-lived stopped R-hadrons decaying out of time with pp collisions using the ATLAS detector. Phys. Rev. D, 88(11), 112003–30pp.
Abstract: An updated search is performed for gluino, top squark, or bottom squark R-hadrons that have come to rest within the ATLAS calorimeter, and decay at some later time to hadronic jets and a neutralino, using 5.0 and 22.9 fb(-1) of pp collisions at 7 and 8 TeV, respectively. Candidate decay events are triggered in selected empty bunch crossings of the LHC in order to remove pp collision backgrounds. Selections based on jet shape and muon system activity are applied to discriminate signal events from cosmic ray and beam-halo muon backgrounds. In the absence of an excess of events, improved limits are set on gluino, stop, and sbottom masses for different decays, lifetimes, and neutralino masses. With a neutralino of mass 100 GeV, the analysis excludes gluinos with mass below 832 GeV (with an expected lower limit of 731 GeV), for a gluino lifetime between 10 μs and 1000 s in the generic R-hadron model with equal branching ratios for decays to q (q) over bar(chi) over tilde (0) and g (chi) over tilde (0). Under the same assumptions for the neutralino mass and squark lifetime, top squarks and bottom squarks in the Regge R-hadron model are excluded with masses below 379 and 344 GeV, respectively.
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Perez, A., & Romanelli, A. (2013). Spatially Dependent Decoherence and Anomalous Diffussion of Quantum Walks. J. Comput. Theor. Nanosci., 10(7), 1591–1595.
Abstract: We analyze the long time behavior of a discrete time quantum walk subject to decoherence with a strong spatial dependence, acting on one half of the lattice. We show that, except for limiting cases on the decoherence parameter, the quantum walk at late times behaves sub-ballistically, meaning that the characteristic features of the quantum walk are not completely spoiled. Contrarily to expectations, the asymptotic behavior is non Markovian, and depends on the amount of decoherence. This feature can be clearly shown on the long time value of the Generalized Chiral Distribution (GCD).
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Hinarejos, M., Bañuls, M. C., & Perez, A. (2013). A Study of Wigner Functions for Discrete-Time Quantum Walks. J. Comput. Theor. Nanosci., 10(7), 1626–1633.
Abstract: We perform a systematic study of the discrete time Quantum Walk on one dimension using Wigner functions, which are generalized to include the chirality (or coin) degree of freedom. In particular, we analyze the evolution of the negative volume in phase space, as a function of time, for different initial states. This negativity can be used to quantify the degree of departure of the system from a classical state. We also relate this quantity to the entanglement between the coin and walker subspaces.
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Sanjuan, R., Nebot, M., Peris, J. B., & Alcami, J. (2013). Immune Activation Promotes Evolutionary Conservation of T-Cell Epitopes in HIV-1. PLoS. Biol., 11(4), e1001523–10pp.
Abstract: The immune system should constitute a strong selective pressure promoting viral genetic diversity and evolution. However, HIV shows lower sequence variability at T-cell epitopes than elsewhere in the genome, in contrast with other human RNA viruses. Here, we propose that epitope conservation is a consequence of the particular interactions established between HIV and the immune system. On one hand, epitope recognition triggers an anti-HIV response mediated by cytotoxic T-lymphocytes (CTLs), but on the other hand, activation of CD4(+) helper T lymphocytes (T-H cells) promotes HIV replication. Mathematical modeling of these opposite selective forces revealed that selection at the intrapatient level can promote either T-cell epitope conservation or escape. We predict greater conservation for epitopes contributing significantly to total immune activation levels (immunodominance), and when T-H cell infection is concomitant to epitope recognition (transinfection). We suggest that HIV-driven immune activation in the lymph nodes during the chronic stage of the disease may offer a favorable scenario for epitope conservation. Our results also support the view that some pathogens draw benefits from the immune response and suggest that vaccination strategies based on conserved T-H epitopes may be counterproductive.
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